Medication Induced Parkinson's Disease

A clinical psychologist brought me in to teach Qigong (Chi Kung) meditation and Tai Chi to her patient group to enhance their sense of well being and provide effective stress management training. So even back then some physicians were seeing the potential Tai Chi offered their clients and even more are now but the number of physicians who are still not informing their patients of Tai Chis direct therapeutic or at the least adjunct therapy benefits to their patients efforts to deal with their conditions and life is increasingly indefensible in this day and age. Medication Induced Parkinson's Disease given the research that has exposed the many physical mental and emotional benefits Tai Chi offers for physicians to not educate themselves on this and share their knowledge with each and every patient is tantamount to mal-practice.

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The new modeling study precisely demonstrates how two -synproteins insert their molecular toes into a neuron's membranepenetrate into it within just a few nanoseconds and immediatelyjoin together as a pair that in itself is not toxic however asmore a-syn proteins join the key threshold is eventuallyoverstepped which results in an accelerated polymerization into aring structure that perforates the membrane which damages thecell. According to Tsigelny it may require many ring structures toactually kill neurons which are generally very durable. Eventhough the nerve cells may be able to repair dozens of ring-inducedperforations and are able to keep the pace with the a-syn assaultup to a certain point they will however at some stage be overtakenby the rate of perforations which results in a gradual appearanceof Parkinson's symptoms that becomes worse. Tsigelny explains: "We think we can create a drug that stops the -syn polymerizationat the point of non-propagating dimmers. By interrupting thepolymerization at this crucial step we may be able to slow thedisease significantly." The experimental validation studies were based on 3-D models ofproteins plus molecular dynamics simulations of the proteinsother modeling techniques and cell-culture experiments. Due to adeeper understanding of -syn polymerization in neurons theresearchers now focus on gaining insight into how monomers of -syn stick to one another. In their pursuit of finding drugcandidates they will include molecules which cause different a-synprotein conformations that are less inclined to stick together asthis effect even if small could decrease symptoms.

The research was published in Current Biology. First author on the study is postdoctoral researcher Yanshan Fangwith additional contributions from postdoctoral researcher LorenaSoares and research technicians Xiuyin Teng and Melissa Geary allof Penn's Department of Biology. When a nerve suffers an acute Medication Induced Parkinson's Disease injury -- as might be caused by apenetrating wound for example or a broken bone that damagesnearby tissues -- the long projection of the nerve cell called theaxon can become injured and degenerate. The process by which itdisintegrates is known as Wallerian or Wallerian-like degenerationand is an active orderly process.